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Catalog no Product name Protocol name IF Magazine title
KTA0004 Annexin V-AbFluor™ 647 Apoptosis Detection kit (Red Fluorescence) Qingda granule attenuates angiotensin II-induced renal apoptosis and activation of the p53 pathway(KTA0004-4.8) 4.8 Frontiers in Endocrinology 
ABP57259 Qingda granule attenuates cardiac fibrosis via suppression of the TGF-β1/Smad2/3 signaling pathway in vitro and in vivo(ABP57259-5) 5 Biomedicine & Pharmacotherapy
A22220 Cy3, Goat Anti-Rabbit IgG Quantitative Proteomics Identified TTC4 as a TBK1 Interactor and a Positive Regulator of SeV-Induced Innate Immunity(A22220-3) 3 Proteomics
BMU106 Qingdi Mixture Alleviates Radiation Enteritis via the PI3K/AKT Signaling Pathway: An Integrated Study Combining Network Pharmacology and Experimental …(BMU106-1.3) 1.3 Animal Bioscience
A23220 Dylight 488, Goat Anti-Rabbit IgG Quantitative Proteomics Identified TTC4 as a TBK1 Interactor and a Positive Regulator of SeV-Induced Innate Immunity(A23220-3) 3 Proteomics
A23220 Dylight 488, Goat Anti-Rabbit IgG Quantitative Proteomics Reveals the Regulatory Networks of Circular RNA CDR1as in Hepatocellular Carcinoma Cells(A23220-4) 4 J Proteome Res
A23210 DyLight 488, Goat Anti-Mouse IgG Quantitative study of the capillaries within the white matter of the Tg2576 mouse model of Alzheimer's disease(A23210-2) 2 Brain Behav
A21010 HRP, Goat Anti-Mouse IgG Quantum dot-labelled antibody based on fluorescence immunoassays for the determination of arsanilic acid in edible pork and liver(A21010-2) 2 Food Addit Contam Part A Chem Anal Control Expo Risk Assess
KTB1600 CheKine™ Micro Reduced Glutathione (GSH) Assay Kit Quercetin Attenuates Acute Kidney Injury Caused by Cisplatin by Inhibiting Ferroptosis and Cuproptosis.(KTB1600-2) 2 Cell Biochemistry and Biophysics
KTP3001 ExKine™ Nuclear and Cytoplasmic Protein Extraction Kit Quercetin alleviates neonatal hypoxic-ischaemic brain injury by rebalancing microglial M1/M2 polarization through silent information regulator 1/high mobility group box-1 signalling.(KTP3001-5.3) 5.3  Inflammopharmacology