Descriptions：表皮生长因子 (EGF) 是 EGF 蛋白家族的成员。EGF 主要由外胚层细胞、单核细胞、肾脏和十二指肠腺分泌。EGF 与其受体 EGFR 结合后，能够刺激上皮细胞生长和增殖，在许多发育过程中发挥重要作用，包括促进牙齿萌出、抑制胃酸分泌以及参与伤口愈合。
Tags：EGF
Descriptions：Fas Ligand (FasL) is a 17.34 kDa tumor necrosis factor with 152 amino acid residues. FasL is expressed from lymphoid tissue and secreted to blood. Binding to its receptor, TNFRSF6/FAS, leads to induce apoptotic signal into cells. Involved in cytotoxic T-cell-mediated apoptosis, natural killer cell-mediated apoptosis and in T-cell development.
Applications：His-SUMO tag
Tags：FasL
Descriptions：Fms相关酪氨酸蛋白酶3配体（Flt-3配体）是由人类FLT3LG基因编码的蛋白质。作为造血的重要调节因子，Flt-3配体酪氨酸蛋白酶具有活性，可刺激淋巴系和髓系造血祖细胞的增殖。Flt-3配体可与其他细胞因子（如GM-CSF、G-CSF、IL-3、EPO、IL-11、IL-12、IL-6或TPO）很好地促进未成熟造血祖细胞的增殖。Flt-3配体在多种造血祖细胞中表达，包括未成熟造血祖细胞和骨髓造血祖细胞。
Tags：Flt-3 Ligand
Descriptions：白细胞介素-10 (IL-10)，细胞因子合成抑制细胞因子(CSIF)，是一种抗炎细胞因子。多种不同类型的产生IL-10，包括免疫细胞和非免疫细胞。IL-10树对突状细胞(DC)和巨噬细胞具有抑制作用，是呈递递的强效制剂，与Th1相关细胞因子IL-2和干扰素-γ(IFN-γ)的产生。IL-10也是感染期间的关键免疫调节剂，因为它能够抑制细胞因子的产生。因此，感染期间过度使用Th1和CD8+ T应答可能会受到抑制。
Tags：IL-10
Descriptions：Interleukin 17F (IL-17F) predicts a molecular mass of 30.1 kDa, is a cytokine of innate and adaptive immune system involved in antimicrobial host defense and maintenance of tissue integrity. It is involved in the development of inflammation and host defense against infection by inducing the expression of genes that encode other proinflammatory cytokines, such as tumor necrosis factor, interleukin 1, interleukin 6 and some members of the colony-stimulating factor family.
Applications：His tag
Tags：IL-17F
Descriptions：Soil N-Acetyl-β-D-glucosaminidase (S-NAG)
Descriptions：Granulocyte-Macrophage Colony Stimulating Factor (GM-CSF) is secreted by a number of different cell types (including activated T cells,Bcells,macrophages,mast cells,endothelial cells and fibroblasts) in response to cytokine or immune and inflammatory stimulation.
Applications：His tag
Tags：M-CSF
Descriptions：EliKine™ Rat GLP-1 Competitive ELISA Kit uses a competitive method to quantify Rat GLP-1 in a sample.
Reactivity：Rat
Applications：ELISA
Tags：No-tag
